The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.

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作者： Sakaki Yumoto M ^[1] ; Kobayashi C ; Sato A ; Fujimura S ; Matsumoto Y ; Takasato M ; Kodama T ; Aburatani H ; Asashima M ; Yoshida N ; Nishinakamura R

作者单位： Division of Integrative Cell Biology, Institute of Molecular Embryology and Genetics, Kumamoto University, Kumamoto 860-0811, Japan. ^[1]

关键词 DNA-Binding Proteins Duane Retraction Syndrome Stem Cells Transcription Factors DNA结合蛋白质类干细胞转录因子

发布时间 2013-11-20

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Development

2006年133卷15期

3005-3013页

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The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.

Development

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Development

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The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.

Development

相似文献

Development

相关期刊

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