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Modeling Alzheimer's disease with iPSCs reveals stress phenotypes associated with intracellular Aβ and differential drug responsiveness

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第一作者: Kondo,T.
作者单位: Department of Neuroanatomy (Biotargeting), Kanazawa University, Graduate School of Medical Sciences [1] Department of Functional Histology, Ehime University, Graduate School of Medicine, Ehime 791-0295 [2] Division of Food Science and Biotechnology, Graduate School of Agriculture, Kyoto University, Kyoto [3] Division of Neurology/Molecular Brain Science, Kobe University, Graduate School of Medicine, Kobe [4] Center for IPS Cell Research and Application (CiRA), Kyoto University, Kyoto 606-8507, Japan, Core [5] Department of Neurology, Graduate School of Medicine, Kyoto University, Kyoto 606-8507, Japan [6] Department of Plastic and Reconstructive Surgery, Graduate School of Medicine, Kyoto University [7] Department of Neurology, Kawasaki Medical School, Okayama 701-0192, Japan [8] Department of Psychiatry, Ehime University, Graduate School of Medicine, Ehime 791-0295, Japan [9] Department of Neurobiology, Northwestern University, Evanston, IL 60208, United States [10] Faculty of Pharmacy and Pharmaceutical Sciences, Fukuyama University, Hiroshima 729-0292, Japan [11] Department of Neuropsychiatry, Institute of Clinical Medicine, University of Tsukuba, Ibaraki 305 [12] Department of Pharmacology, Faculty of Medicine, Saitama Medical University, Saitama 350-0495, Japan [13] Center for IPS Cell Research and Application (CiRA), Kyoto University, Kyoto 606-8507, Japan [14] Department of Neuroscience, Graduate School of Medicine, Osaka City University, Osaka 545-8585 [15] Department of Biological Sciences, Graduate School of Medicine, Kyoto University, Kyoto 606-8507 [16] Core Research for Evolutional Science and Technology (CREST), Japan Science and Technology Agency [17] Department of Morphological Brain Science, Graduate School of Medicine, Kyoto University, Kyoto 606 [18]
发布时间 2013-11-20
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