Protein kinase- and lipase inhibitors of inositide metabolism deplete IP <ce:inf loc="post">7</ce:inf> indirectly in pancreatic β-cells: Off-target effects on cellular bioenergetics and direct effects on IP6K activity

导出原文传递

翻译打印收藏纠错

作者： Subu Surendran Rajasekaran ^[1] ; Christopher Illies ^[2] ; Stephen B. Shears ^[3] ; Huanchen Wang ; Thais S. Ayala ; Joilson O. Martins ; Elisabetta Daré ; Per-Olof Berggren ; Christopher J. Barker

作者单位： Laboratory of Immunoendocrinology, Department of Clinical and Toxicological Analyses, Faculty of Pharmaceutical Sciences, University of S?o Paulo ^[1] Signal Transduction Laboratory/Inositol Signaling Group, NIEHS ^[2] The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet ^[3]

关键词 Di-phosphoinositol pentakisphosphate Inositol pyrophosphate Inositol hexakisphosphate kinase ATP/ADP Pancreatic β-cell Pharmacological inhibitors

发布时间 2018-06-23

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Cellular Signalling

2018年42卷

127-133页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Protein kinase- and lipase inhibitors of inositide metabolism deplete IP <ce:inf loc="post">7</ce:inf> indirectly in pancreatic β-cells: Off-target effects on cellular bioenergetics and direct effects on IP6K activity

Cellular Signalling

相似文献

Cellular Signalling

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Protein kinase- and lipase inhibitors of inositide metabolism deplete IP <ce:inf loc="post">7</ce:inf> indirectly in pancreatic β-cells: Off-target effects on cellular bioenergetics and direct effects on IP6K activity

Cellular Signalling

相似文献

Cellular Signalling

相关期刊

检索历史清除