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Induction of beta-cell rest by a Kir6.2/SUR1-selective K(ATP)-channel opener preserves beta-cell insulin stores and insulin secretion in human islets cultured at high (11 mM) glucose.

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第一作者： Ritzel RA

作者： Ritzel RA ^[1] ; Hansen JB ; Veldhuis JD ; Butler PC

作者单位： Division of Endocrinology and Diabetes, Keck School of Medicine, University of Southern California, Los Angeles, California 90033, USA. ^[1]

关键词 Adenosine Triphosphate Bicyclo Compounds Heterocyclic Cyclic S-Oxides 腺苷三磷酸二环化合物杂环环硫氧化物葡萄糖胰岛素胰岛钾通道

发布时间 2013-11-20

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The Journal of Clinical Endocrinology and Metabolism

2004年89卷2期

795-805页

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The Journal of Clinical Endocrinology and Metabolism

2004年89卷2期

795-805页

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Induction of beta-cell rest by a Kir6.2/SUR1-selective K(ATP)-channel opener preserves beta-cell insulin stores and insulin secretion in human islets cultured at high (11 mM) glucose.

The Journal of Clinical Endocrinology and Metabolism

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The Journal of Clinical Endocrinology and Metabolism

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Induction of beta-cell rest by a Kir6.2/SUR1-selective K(ATP)-channel opener preserves beta-cell insulin stores and insulin secretion in human islets cultured at high (11 mM) glucose.

The Journal of Clinical Endocrinology and Metabolism

相似文献

同项目论文

The Journal of Clinical Endocrinology and Metabolism

相关期刊

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