Inhibition of transforming growth factor beta1-induced hepatoma cell apoptosis by liver tumor promoters: characterization of primary signaling events and effects on CPP32-like caspase activity.
第一作者:
A,Buchmann
第一单位:
Institute of Toxicology, University of Tübingen, Wilhelmstr. 56, 72074 Tübingen, Germany. ALBRECHT.BUCHMANN@UNI-TUEBINGEN.DE
作者:
医学主题词
动物(Animals);细胞凋亡(Apoptosis);钙-钙调素依赖性蛋白激酶类(Calcium-Calmodulin-Dependent Protein Kinases);半胱氨酸天冬氨酸蛋白酶3(Caspase 3);半胱氨酸天冬氨酸蛋白酶(Caspases);氯贝丁酯(Clofibrate);滴滴涕(DDT);地塞米松(Dexamethasone);狄氏剂(Dieldrin);胰岛素(Insulin);肝肿瘤, 实验性(Liver Neoplasms, Experimental);NF-κB(NF-kappa B);苯巴比妥(Phenobarbital);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);大鼠(Rats);信号传导(Signal Transduction);时间因素(Time Factors);转化生长因子β(Transforming Growth Factor beta);肿瘤细胞, 培养的(Tumor Cells, Cultured);bcl-2相关X蛋白质(bcl-2-Associated X Protein);bcl-X蛋白质(bcl-X Protein)
DOI
10.1038/sj.cdd.4400475
PMID
10200566
发布时间
2021-12-03
- 浏览9
Cell death and differentiation
190-200页
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