Transgenic mice expressing a Huntington's disease mutation are resistant to quinolinic acid-induced striatal excitotoxicity.
第一作者:
O,Hansson
第一单位:
Section for Neuronal Survival, Wallenberg Neuroscience Center, Department of Physiological Sciences, Lund University, Sölvegatan 17, S-223 62 Lund, Sweden. oskar.hansson@mphy.lu.se
作者:
主题词
动物(Animals);脑(Brain);细胞计数(Cell Count);细胞存活(Cell Survival);纹状体(Corpus Striatum);外显子(Exons);人类(Humans);杭廷顿病(Huntington Disease);免疫组织化学(Immunohistochemistry);原位缺口末端标记(In Situ Nick-End Labeling);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);N-甲基天冬氨酸(N-Methylaspartate);神经组织蛋白质类(Nerve Tissue Proteins);神经毒素类(Neurotoxins);喹啉酸(Quinolinic Acid);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate)
DOI
10.1073/pnas.96.15.8727
PMID
10411943
发布时间
2019-05-01
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