换一批Insulin inhibits the activation of transcription by a C-terminal fragment of the forkhead transcription factor FKHR. A mechanism for insulin inhibition of insulin-like growth factor-binding protein-1 transcription.
第一作者：
M,Tomizawa
第一单位：
Growth and Development Section, Clinical Endocrinology Branch, NIDDK, National Institutes of Health, Bethesda, Maryland 20892, USA.
作者：
医学主题词
动物(Animals);DNA结合蛋白质类(DNA-Binding Proteins);叉头转录因子类(Forkhead Transcription Factors);胰岛素(Insulin);胰岛素样生长因子结合蛋白质1(Insulin-Like Growth Factor Binding Protein 1);小鼠(Mice);神经组织蛋白质类(Nerve Tissue Proteins);肽碎片(Peptide Fragments);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);启动区, 遗传(Promoter Regions, Genetic);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);RNA, 信使(RNA, Messenger);大鼠(Rats);转录因子(Transcription Factors);转录激活(Transcriptional Activation)
DOI
10.1074/jbc.275.10.7289
PMID
10702299
发布时间
2022-12-07
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