Recombinant human interleukin-11 prevents mucosal atrophy and bowel shortening in the defunctionalized intestine.

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作者： E C,Dickinson ^[1] ; R,Tuncer ; E P,Nadler ; U,Koltuksuz ; P,Boyle ; S M,Alber ; S C,Watkins ; H R,Ford

作者单位： Department of Surgery, Children's Hospital of Pittsburgh, University of Pittsburgh School of Medicine and the Center for Biological Imaging, Pennsylvania, USA. ^[1]

主题词动物(Animals);萎缩(Atrophy);人类(Humans);白细胞介素11(Interleukin-11);肠黏膜(Intestinal Mucosa);男(雄)性(Male);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);重组蛋白质类(Recombinant Proteins);短肠综合征(Short Bowel Syndrome)

DOI 10.1053/jpsu.2000.7826

PMID 10917300

发布时间 2007-11-14

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Journal of pediatric surgery

2000年35卷7期

1079-83页

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Recombinant human interleukin-11 prevents mucosal atrophy and bowel shortening in the defunctionalized intestine.

Journal of pediatric surgery

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Journal of pediatric surgery

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Recombinant human interleukin-11 prevents mucosal atrophy and bowel shortening in the defunctionalized intestine.

Journal of pediatric surgery

相似文献

Journal of pediatric surgery

相关期刊

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