Antioxidants inhibit JNK and p38 MAPK activation but not ERK 1/2 activation by angiotensin II in rat aortic smooth muscle cells.
第一作者:
M,Kyaw
第一单位:
Department of Pharmacology, The University of Tokushima School of Medicine, Japan.
作者:
医学主题词
乙酰半胱氨酸(Acetylcysteine);血管紧张素Ⅱ(Angiotensin II);动物(Animals);抗氧化剂(Antioxidants);主动脉, 胸(Aorta, Thoracic);抗坏血酸(Ascorbic Acid);细胞, 培养的(Cells, Cultured);螯合剂(Chelating Agents);色满类(Chromans);二硫代氨基甲酸酯(Ditiocarb);剂量效应关系, 药物(Dose-Response Relationship, Drug);酶抑制剂(Enzyme Inhibitors);自由基清除剂(Free Radical Scavengers);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);MAP激酶激酶4(MAP Kinase Kinase 4);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);丝裂原激活蛋白激酶激酶类(Mitogen-Activated Protein Kinase Kinases);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);肌, 平滑, 血管(Muscle, Smooth, Vascular);鎓类化合物(Onium Compounds);氧耗量(Oxygen Consumption);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);活性氧(Reactive Oxygen Species);血管收缩药(Vasoconstrictor Agents);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1291/hypres.24.251
PMID
11409648
发布时间
2019-10-25
- 浏览18
Hypertension research
251-61页
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