Glutathione levels and BAX activation during apoptosis due to oxidative stress in cells expressing wild-type and mutant cystic fibrosis transmembrane conductance regulator.
作者:
主题词
氨基酸取代(Amino Acid Substitution);细胞凋亡(Apoptosis);丁硫氨酸硫酸亚胺(Buthionine Sulfoximine);囊性纤维化跨膜传导调节因子(Cystic Fibrosis Transmembrane Conductance Regulator);谷胱甘肽(Glutathione);人类(Humans);过氧化氢(Hydrogen Peroxide);动力学(Kinetics);诱变(Mutagenesis);氧化性应激(Oxidative Stress);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);呼吸道黏膜(Respiratory Mucosa);转染(Transfection);bcl-2相关X蛋白质(bcl-2-Associated X Protein)
DOI
10.1074/jbc.M110288200
PMID
12023951
发布时间
2021-02-06
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