换一批Spinal noradrenergic activation mediates allodynia reduction from an allosteric adenosine modulator in a rat model of neuropathic pain.
第一作者：
Xinhui,Li
第一单位：
Department of Anesthesiology and Center for the Study of Pharmacologic Plasticity in the Presence of Pain, Wake Forest University School of Medicine, Medical Center Boulevard, Winston-Salem, NC 27157-1009, USA. xli@wfubmc.edu
作者：
医学主题词
腺苷(Adenosine);肾上腺素能α拮抗剂(Adrenergic alpha-Antagonists);别构调节(Allosteric Regulation);动物(Animals);多巴胺β羟化酶(Dopamine beta-Hydroxylase);咪唑克生(Idazoxan);免疫毒素类(Immunotoxins);结扎术(Ligation);男(雄)性(Male);N-糖基水解酶类(N-Glycosyl Hydrolases);神经痛(Neuralgia);去甲肾上腺素(Norepinephrine);痛阈(Pain Threshold);物理刺激(Physical Stimulation);植物蛋白质类(Plant Proteins);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 肾上腺素能α2(Receptors, Adrenergic, alpha-2);核糖体失活蛋白质类, 1型(Ribosome Inactivating Proteins, Type 1);脊髓(Spinal Cord);脊神经(Spinal Nerves);可可碱(Theobromine);黄嘌呤类(Xanthines)
DOI
10.1016/s0304-3959(02)00011-8
PMID
12031785
发布时间
2019-08-18
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Pain
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