Contributions of mitogen-activated protein kinase and nuclear factor kappa B to N-(4-hydroxyphenyl)retinamide-induced apoptosis in prostate cancer cells.
作者:
主题词
抗肿瘤药(Antineoplastic Agents);细胞凋亡(Apoptosis);半胱氨酸天冬氨酸蛋白酶3(Caspase 3);半胱氨酸天冬氨酸蛋白酶(Caspases);细胞存活(Cell Survival);抗药性, 肿瘤(Drug Resistance, Neoplasm);电泳迁移位移试验(Electrophoretic Mobility Shift Assay);芬维A铵(Fenretinide);人类(Humans);I-κB激酶(I-kappa B Kinase);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);MAP激酶激酶4(MAP Kinase Kinase 4);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);丝裂原活化蛋白激酶6(Mitogen-Activated Protein Kinase 6);丝裂原激活蛋白激酶激酶类(Mitogen-Activated Protein Kinase Kinases);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);NF-κB(NF-kappa B);腈类(Nitriles);有机化学品(Organic Chemicals);前列腺肿瘤(Prostatic Neoplasms);砜类(Sulfones);肿瘤细胞, 培养的(Tumor Cells, Cultured);β半乳糖苷酶类(beta-Galactosidase)
DOI
10.1002/mc.10084
PMID
12410564
发布时间
2021-12-03
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Molecular carcinogenesis
127-37页
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