换一批Growth factor independence-1B expression leads to defects in T cell activation, IL-7 receptor alpha expression, and T cell lineage commitment.
第一作者：
Loretta L,Doan
第一单位：
Institute for Cellular Therapeutics and Department of Surgery, University of Louisville School of Medicine, Louisville, KY 40202, USA.
作者：
医学主题词
动物(Animals);自身抗原(Autoantigens);细胞分化(Cell Differentiation);细胞谱系(Cell Lineage);交联试剂(Cross-Linking Reagents);DNA结合蛋白质类(DNA-Binding Proteins);女(雌)性(Female);基因表达调控(Gene Expression Regulation);基因, MHCⅠ类(Genes, MHC Class I);H-Y抗原(H-Y Antigen);白细胞介素2(Interleukin-2);淋巴细胞减少(Lymphopenia);淋巴细胞生成(Lymphopoiesis);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);受体, 抗原, T细胞, α-β(Receptors, Antigen, T-Cell, alpha-beta);受体, 白细胞介素7(Receptors, Interleukin-7);阻遏蛋白质类(Repressor Proteins);T淋巴细胞亚群(T-Lymphocyte Subsets);胸腺(Thymus Gland);转录因子(Transcription Factors);转基因(Transgenes)
DOI
10.4049/jimmunol.170.5.2356
PMID
12594258
发布时间
2019-05-16
- 浏览48
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
