[K(+)](o)-dependent change in conformation of the HERG1 long QT mutation N629D channel results in partial reversal of the in vitro disease phenotype.
第一作者:
Guo Qi,Teng
第一单位:
Department of Medicine, University of Calgary, Alberta, Canada.
作者:
医学主题词
动物(Animals);阳离子转运蛋白质类(Cation Transport Proteins);电生理学(Electrophysiology);快速延迟整流钾通道(Ether-A-Go-Go Potassium Channels);女(雌)性(Female);QT延长综合征(Long QT Syndrome);突变(Mutation);卵母细胞(Oocytes);膜片钳术(Patch-Clamp Techniques);表型(Phenotype);钾(Potassium);钾通道(Potassium Channels);钾通道, 电压门控(Potassium Channels, Voltage-Gated);爪蟾属(Xenopus)
DOI
10.1016/s0008-6363(02)00778-2
PMID
12618226
发布时间
2019-05-13
- 浏览2
Cardiovascular research
642-50页
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