Sialylation and sulfation of lactosylceramide distinctly regulate anchorage-independent growth, apoptosis, and gene expression in 3LL Lewis lung carcinoma cells.

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作者： Satoshi,Uemura ^[1] ; Kazuya,Kabayama ; Mariko,Noguchi ; Yasuyuki,Igarashi ; Jin-Ichi,Inokuchi

作者单位： Department of Biomembrane and Biofunctional Chemistry, Graduate School of Pharmaceutical Sciences, Hokkaido University, Kita 12-jo, Nishi 6-chome, Kita-ku, Sapporo 060-0812, Japan. ^[1]

主题词动物(Animals);抗原, CD(Antigens, CD);细胞凋亡(Apoptosis);癌, Lewis肺(Carcinoma, Lewis Lung);细胞黏附(Cell Adhesion);细胞分裂(Cell Division);细胞转化, 肿瘤(Cell Transformation, Neoplastic);酶激活(Enzyme Activation);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);免疫球蛋白Gm同种异型(Immunoglobulin Gm Allotypes);乳酸酰基鞘氨醇类(Lactosylceramides);小鼠(Mice);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);N-乙酰神经氨酸(N-Acetylneuraminic Acid);血小板源性生长因子(Platelet-Derived Growth Factor);受体, 血小板源生长因子α(Receptor, Platelet-Derived Growth Factor alpha);硫酸盐类(Sulfates);肿瘤细胞, 培养的(Tumor Cells, Cultured)

DOI 10.1093/glycob/cwg022

PMID 12626418

发布时间 2017-11-16

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Glycobiology

2003年13卷3期

207-16页

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Sialylation and sulfation of lactosylceramide distinctly regulate anchorage-independent growth, apoptosis, and gene expression in 3LL Lewis lung carcinoma cells.

Glycobiology

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Glycobiology

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Sialylation and sulfation of lactosylceramide distinctly regulate anchorage-independent growth, apoptosis, and gene expression in 3LL Lewis lung carcinoma cells.

Glycobiology

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Glycobiology

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