换一批
换一批c-Jun NH2-terminal kinase-dependent Fas activation contributes to etoposide-induced apoptosis in p53-mutated prostate cancer cells.
作者：
主题词
抗肿瘤药, 植物(Antineoplastic Agents, Phytogenic);细胞凋亡(Apoptosis);拟南芥蛋白质类(Arabidopsis Proteins);印迹法, 蛋白质(Blotting, Western);半胱氨酸天冬氨酸蛋白酶(Caspases);DNA碎片裂(DNA Fragmentation);依托泊甙(Etoposide);脂肪酸去饱和酶类(Fatty Acid Desaturases);流式细胞术(Flow Cytometry);基因, p53(Genes, p53);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);MAP激酶激酶7(MAP Kinase Kinase 7);男(雄)性(Male);丝裂原激活蛋白激酶激酶类(Mitogen-Activated Protein Kinase Kinases);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);突变(Mutation);前列腺肿瘤(Prostatic Neoplasms);蛋白质类(Proteins);转染(Transfection);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1002/pros.10227
PMID
12712406
发布时间
2017-11-16
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The Prostate
265-80页
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