换一批IL-17 production from activated T cells is required for the spontaneous development of destructive arthritis in mice deficient in IL-1 receptor antagonist.
第一作者：
Susumu,Nakae
第一单位：
Center for Experimental Medicine and Department of Cancer Biology, Institute of Medical Science, University of Tokyo, 4-6-1 Shirokanedai, Minato-ku, Tokyo 108-8639, Japan.
作者：
医学主题词
动物(Animals);关节炎, 类风湿(Arthritis, Rheumatoid);CD40配体(CD40 Ligand);交叉, 遗传(Crosses, Genetic);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);基因表达调控(Gene Expression Regulation);白细胞介素1受体拮抗蛋白质(Interleukin 1 Receptor Antagonist Protein);白细胞介素1(Interleukin-1);白细胞介素17(Interleukin-17);淋巴细胞活化(Lymphocyte Activation);男(雄)性(Male);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);小鼠, 基因敲除(Mice, Knockout);OX40配体(OX40 Ligand);唾液糖蛋白类(Sialoglycoproteins);T淋巴细胞(T-Lymphocytes);肿瘤坏死因子类(Tumor Necrosis Factors)
DOI
10.1073/pnas.1035999100
PMID
12721360
发布时间
2022-03-18
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