Interleukin-6 differentially regulates androgen receptor transactivation via PI3K-Akt, STAT3, and MAPK, three distinct signal pathways in prostate cancer cells.
作者:
主题词
DNA结合蛋白质类(DNA-Binding Proteins);酶抑制剂(Enzyme Inhibitors);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);白细胞介素6(Interleukin-6);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);乳房瘤病毒, 鼠(Mammary Tumor Virus, Mouse);模型, 生物学(Models, Biological);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);启动区, 遗传(Promoter Regions, Genetic);前列腺肿瘤(Prostatic Neoplasms);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);受体, 雄激素(Receptors, Androgen);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction);反式激活因子类(Trans-Activators);转录激活(Transcriptional Activation);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1016/s0006-291x(03)00792-7
PMID
12763015
发布时间
2021-12-03
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