Antitumor efficacy of intermittent treatment schedules with the rapamycin derivative RAD001 correlates with prolonged inactivation of ribosomal protein S6 kinase 1 in peripheral blood mononuclear cells.

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作者： Anne,Boulay ^[1] ; Sabine,Zumstein-Mecker ; Christine,Stephan ; Iwan,Beuvink ; Frederic,Zilbermann ; Roland,Haller ; Sonja,Tobler ; Christoph,Heusser ; Terence,O'Reilly ; Barbara,Stolz ; Andreas,Marti ; George,Thomas ; Heidi A,Lane

作者单位： Novartis Institutes for BioMedical Research Basel, Novartis Pharma AG, Basel, Switzerland. ^[1]

主题词动物(Animals);疾病模型, 动物(Disease Models, Animal);剂量效应关系, 药物(Dose-Response Relationship, Drug);用药计划表(Drug Administration Schedule);药物监测(Drug Monitoring);人类(Humans);免疫抑制剂(Immunosuppressive Agents);白细胞, 单核(Leukocytes, Mononuclear);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);胰腺肿瘤(Pancreatic Neoplasms);大鼠(Rats);大鼠, 近交Lew(Rats, Inbred Lew);核糖体蛋白质S6激酶类, 90-kDa(Ribosomal Protein S6 Kinases, 90-kDa);西罗莫司(Sirolimus);时间因素(Time Factors);肿瘤细胞, 培养的(Tumor Cells, Cultured)

DOI 10.1158/0008-5472.can-3554-2

PMID 14729632

发布时间 2022-03-30

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Cancer research

2004年64卷1期

252-61页

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Antitumor efficacy of intermittent treatment schedules with the rapamycin derivative RAD001 correlates with prolonged inactivation of ribosomal protein S6 kinase 1 in peripheral blood mononuclear cells.

Cancer research

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Cancer research

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Antitumor efficacy of intermittent treatment schedules with the rapamycin derivative RAD001 correlates with prolonged inactivation of ribosomal protein S6 kinase 1 in peripheral blood mononuclear cells.

Cancer research

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Cancer research

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