换一批
换一批Down-regulation of nuclear protein ICBP90 by p53/p21Cip1/WAF1-dependent DNA-damage checkpoint signals contributes to cell cycle arrest at G1/S transition.
第一作者：
Yoshimi,Arima
第一单位：
Department of Tumour Genetics and Biology, Graduate School of Medical Sciences, Kumamoto University, Kumamoto 860-8556, Japan.
作者：
主题词
鹅膏菌素类(Amanitins);抗原, 肿瘤(Antigens, Neoplasm);CCAAT增强子结合蛋白类(CCAAT-Enhancer-Binding Proteins);咖啡因(Caffeine);细胞周期蛋白B(Cyclin B);周期素依赖激酶抑制剂p21(Cyclin-Dependent Kinase Inhibitor p21);细胞周期蛋白类(Cyclins);DNA损伤(DNA Damage);DNA拓扑异构酶类, Ⅱ型(DNA Topoisomerases, Type II);DNA结合蛋白质类(DNA-Binding Proteins);减量调节(Down-Regulation);多柔比星(Doxorubicin);流式细胞术(Flow Cytometry);G1期(G1 Phase);基因表达(Gene Expression);基因, cdc(Genes, cdc);HCT116细胞(HCT116 Cells);组蛋白类(Histones);人类(Humans);显微镜检查, 荧光(Microscopy, Fluorescence);RNA, 小分子干扰(RNA, Small Interfering);S期(S Phase);信号传导(Signal Transduction);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53);泛素蛋白连接酶类(Ubiquitin-Protein Ligases)
DOI
10.1111/j.1356-9597.2004.00710.x
PMID
15009091
发布时间
2020-12-08
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Genes to cells
131-42页
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