换一批
换一批Uteroplacental insufficiency alters DNA methylation, one-carbon metabolism, and histone acetylation in IUGR rats.
第一作者：
Nicole K,MacLennan
第一单位：
David Geffen School of Medicine, UCLA, Department of Pediatrics, Division of Neonatology and Developmental Biology, Mattel Children's Hospital, UCLA, Los Angeles, California, 90095-1752, USA.
作者：
主题词
乙酰化作用(Acetylation);动物(Animals);碳(Carbon);染色质(Chromatin);胱硫醚β合酶(Cystathionine beta-Synthase);DNA甲基化(DNA Methylation);疾病模型, 动物(Disease Models, Animal);疾病易感性(Disease Susceptibility);酶诱导(Enzyme Induction);女(雌)性(Female);胎儿生长迟缓(Fetal Growth Retardation);基因表达调控, 发育期(Gene Expression Regulation, Developmental);孕龄(Gestational Age);组蛋白类(Histones);肝(Liver);甲硫氨酸(Methionine);甲硫氨酸腺苷转移酶(Methionine Adenosyltransferase);胎盘血液循环(Placental Circulation);胎盘功能不全(Placental Insufficiency);妊娠(Pregnancy);蛋白质加工, 转译后(Protein Processing, Post-Translational);大鼠(Rats);S-腺苷高半胱氨酸(S-Adenosylhomocysteine)
DOI
10.1152/physiolgenomics.00042.2004
PMID
15084713
发布时间
2013-11-21
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Physiological genomics
43-50页
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