AT1A-mediated activation of kidney JNK1 and SMAD2 in obstructive uropathy: preservation of kidney tissue mass using candesartan.
第一作者:
Ann,Wamsley-Davis
第一单位:
Renal Section, Department of Medicine, Baylor College of Medicine, Houston, Texas 77030, USA.
作者:
主题词
动物(Animals);抗高血压药(Antihypertensive Agents);苯咪唑类(Benzimidazoles);联苯化合物(Biphenyl Compounds);DNA结合蛋白质类(DNA-Binding Proteins);减量调节(Down-Regulation);男(雄)性(Male);丝裂原活化蛋白激酶8(Mitogen-Activated Protein Kinase 8);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 血管紧张素, 1型(Receptor, Angiotensin, Type 1);Smad2蛋白质(Smad2 Protein);四唑类(Tetrazoles);反式激活因子类(Trans-Activators);增量调节(Up-Regulation);输尿管梗阻(Ureteral Obstruction)
DOI
10.1152/ajprenal.00452.2003
PMID
15126250
发布时间
2021-12-03
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