Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalis.

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作者： Ozlem,Yilmaz ^[1] ; Thomas,Jungas ; Philippe,Verbeke ; David M,Ojcius

作者单位： Department of Pathobiology, University of Washington, School of Public Health, HSC Box 357238, Seattle, WA 98195, USA. ozlem@u.washington.edu ^[1]

主题词细胞凋亡(Apoptosis);拟杆菌科感染(Bacteroidaceae Infections);细胞色素c类(Cytochromes c);DNA碎片裂(DNA Fragmentation);上皮细胞(Epithelial Cells);人类(Humans);线粒体(Mitochondria);牙周疾病(Periodontal Diseases);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);龈紫单胞菌(Porphyromonas gingivalis);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);信号传导(Signal Transduction)

DOI 10.1128/IAI.72.7.3743-3751.2004

PMID 15213114

发布时间 2021-12-03

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Infection and immunity

2004年72卷7期

3743-51页

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Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalis.

Infection and immunity

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Infection and immunity

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Activation of the phosphatidylinositol 3-kinase/Akt pathway contributes to survival of primary epithelial cells infected with the periodontal pathogen Porphyromonas gingivalis.

Infection and immunity

相似文献

Infection and immunity

相关期刊

检索历史清除