ALS2CL, the novel protein highly homologous to the carboxy-terminal half of ALS2, binds to Rab5 and modulates endosome dynamics.

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作者： Shinji,Hadano ^[1] ; Asako,Otomo ; Kyoko,Suzuki-Utsunomiya ; Ryota,Kunita ; Yoshiko,Yanagisawa ; Junko,Showguchi-Miyata ; Hikaru,Mizumura ; Joh-E,Ikeda

作者单位： Department of Molecular Neuroscience, The Institute of Medical Sciences, Tokai University, Isehara, Kanagawa 259-1193, Japan. ^[1]

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);选择性剪接(Alternative Splicing);氨基酸序列(Amino Acid Sequence);肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);载体蛋白质类(Carrier Proteins);内颗粒(Endosomes);鸟嘌呤核苷酸交换因子类(Guanine Nucleotide Exchange Factors);人类(Humans);小鼠(Mice);分子序列数据(Molecular Sequence Data);蛋白质结合(Protein Binding);蛋白质结构, 三级(Protein Structure, Tertiary);序列比对(Sequence Alignment);亚细胞部分(Subcellular Fractions);组织分布(Tissue Distribution);rab5 GTP结合蛋白质类(rab5 GTP-Binding Proteins)

DOI 10.1016/j.febslet.2004.07.092

PMID 15388334

发布时间 2006-11-15

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2004年575卷1-3期

64-70页

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ALS2CL, the novel protein highly homologous to the carboxy-terminal half of ALS2, binds to Rab5 and modulates endosome dynamics.

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ALS2CL, the novel protein highly homologous to the carboxy-terminal half of ALS2, binds to Rab5 and modulates endosome dynamics.

FEBS letters

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FEBS letters

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