换一批
换一批Skeletal defects in ringelschwanz mutant mice reveal that Lrp6 is required for proper somitogenesis and osteogenesis.
作者：
主题词
衰老(Aging);等位基因(Alleles);氨基酸序列(Amino Acid Sequence);动物(Animals);碱基序列(Base Sequence);碱性螺旋-环-螺旋转录因子类(Basic Helix-Loop-Helix Transcription Factors);躯体发育模式(Body Patterning);细胞极性(Cell Polarity);细胞支架蛋白质类(Cytoskeletal Proteins);DNA结合蛋白质类(DNA-Binding Proteins);胚胎, 哺乳动物(Embryo, Mammalian);成纤维细胞(Fibroblasts);腰骶部(Lumbosacral Region);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);分子序列数据(Molecular Sequence Data);肌肉骨骼畸形(Musculoskeletal Abnormalities);突变(Mutation);骨生成(Osteogenesis);表型(Phenotype);原癌基因蛋白质类(Proto-Oncogene Proteins);受体, LDL(Receptors, LDL);序列比对(Sequence Alignment);信号传导(Signal Transduction);体节(Somites);反式激活因子类(Trans-Activators);Wnt蛋白质类(Wnt Proteins);β连环素(beta Catenin)
DOI
10.1242/dev.01405
PMID
15469977
发布时间
2022-02-24
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