换一批Identification of mcl-1 as a BCR/ABL-dependent target in chronic myeloid leukemia (CML): evidence for cooperative antileukemic effects of imatinib and mcl-1 antisense oligonucleotides.
第一作者：
Karl J,Aichberger
第一单位：
Department of Internal Medicine I, Division of Hematology and Hemostaseology, Medical University of Vienna, AKH-Wien, Waehringer Guertel 18-20, A-1097 Vienna, Austria.
作者：
主题词
动物(Animals);抗肿瘤药(Antineoplastic Agents);苯甲酰胺类(Benzamides);细胞存活(Cell Survival);DNA结合蛋白质类(DNA-Binding Proteins);减量调节(Down-Regulation);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);融合蛋白质类, bcr-abl(Fusion Proteins, bcr-abl);基因表达调控, 白血病(Gene Expression Regulation, Leukemic);人类(Humans);K562细胞(K562 Cells);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);MAP激酶激酶激酶类(MAP Kinase Kinase Kinases);乳蛋白质类(Milk Proteins);肿瘤蛋白质类(Neoplasm Proteins);寡核苷酸类, 反义(Oligonucleotides, Antisense);哌嗪类(Piperazines);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);嘧啶类(Pyrimidines);RNA, 小分子干扰(RNA, Small Interfering);STAT5转录因子(STAT5 Transcription Factor);反式激活因子类(Trans-Activators);肿瘤细胞, 培养的(Tumor Cells, Cultured);raf激酶类(raf Kinases);ras蛋白质类(ras Proteins)
DOI
10.1182/blood-2004-02-0749
PMID
15626746
发布时间
2021-02-06
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