Anti-IL-16 therapy reduces CD4+ T-cell infiltration and improves paralysis and histopathology of relapsing EAE.
第一作者:
Dusanka S,Skundric
第一单位:
Department of Neurology, Wayne State University School of Medicine, Detroit, Michigan 48201, USA. skundric@cmb.biosci.wayne.edu
作者:
主题词
动物(Animals);抗体(Antibodies);B-淋巴细胞(B-Lymphocytes);印迹法, 蛋白质(Blotting, Western);CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);疾病模型, 动物(Disease Models, Animal);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);女(雌)性(Female);糖蛋白类(Glycoproteins);免疫法(Immunization);免疫组织化学(Immunohistochemistry);免疫疗法(Immunotherapy);吲哚类(Indoles);白细胞介素16(Interleukin-16);淋巴细胞活化(Lymphocyte Activation);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);显微镜检查, 电子, 透射(Microscopy, Electron, Transmission);多发性硬化, 复发缓解性(Multiple Sclerosis, Relapsing-Remitting);麻痹(Paralysis);肽碎片(Peptide Fragments);表型(Phenotype);反应时间(Reaction Time);疾病严重程度指数(Severity of Illness Index);脊髓(Spinal Cord);染色与标记(Staining and Labeling);时间因素(Time Factors)
DOI
10.1002/jnr.20377
PMID
15682385
发布时间
2017-11-16
- 浏览12
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文