Loss of mitogen-activated protein kinase kinase kinase 4 (MEKK4) results in enhanced apoptosis and defective neural tube development.

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作者： Hongbo,Chi ^[1] ; Matthew R,Sarkisian ; Pasko,Rakic ; Richard A,Flavell

作者单位： Section of Immunobiology, Department of Neurobiology and Kavli Institute of Neuroscience, and Howard Hughes Medical Institute, Yale University School of Medicine, New Haven, CT 06520, USA. ^[1]

主题词动物(Animals);细胞凋亡(Apoptosis);脑(Brain);MAP激酶激酶4(MAP Kinase Kinase 4);MAP激酶激酶激酶4(MAP Kinase Kinase Kinase 4);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);丝裂原活化蛋白激酶8(Mitogen-Activated Protein Kinase 8);神经管缺损(Neural Tube Defects)

DOI 10.1073/pnas.0500026102

PMID 15731347

发布时间 2022-03-09

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Proceedings of the National Academy of Sciences of the United States of America

2005年102卷10期

3846-51页

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Loss of mitogen-activated protein kinase kinase kinase 4 (MEKK4) results in enhanced apoptosis and defective neural tube development.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

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Loss of mitogen-activated protein kinase kinase kinase 4 (MEKK4) results in enhanced apoptosis and defective neural tube development.

Proceedings of the National Academy of Sciences of the United States of America

相似文献

Proceedings of the National Academy of Sciences of the United States of America

相关期刊

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