换一批
换一批Heat shock protein 60 inhibits Th1-mediated hepatitis model via innate regulation of Th1/Th2 transcription factors and cytokines.
第一作者：
Alexandra,Zanin-Zhorov
第一单位：
Department of Immunology, Weizmann Institute of Science, Rehovot, Israel.
作者：
主题词
主动转运, 细胞核(Active Transport, Cell Nucleus);动物(Animals);伴侣蛋白60(Chaperonin 60);DNA结合蛋白质类(DNA-Binding Proteins);女(雌)性(Female);GATA3转录因子(GATA3 Transcription Factor);肝炎, 动物(Hepatitis, Animal);人类(Humans);免疫, 先天(Immunity, Innate);干扰素γ(Interferon-gamma);白细胞介素10(Interleukin-10);淋巴细胞活化(Lymphocyte Activation);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);NF-κB(NF-kappa B);NFATC转录因子类(NFATC Transcription Factors);核蛋白质类(Nuclear Proteins);受体, 细胞表面(Receptors, Cell Surface);T盒域蛋白质类(T-Box Domain Proteins);Th1细胞(Th1 Cells);Th2细胞(Th2 Cells);Toll样受体2(Toll-Like Receptor 2);Toll样受体(Toll-Like Receptors);反式激活因子类(Trans-Activators);转录因子(Transcription Factors);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.4049/jimmunol.174.6.3227
PMID
15749853
发布时间
2025-01-03
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