5-HT2A/2C receptor signaling via phospholipase A2 and arachidonic acid is attenuated in mice lacking the serotonin reuptake transporter.

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作者： Ying,Qu ^[1] ; Nelly,Villacreses ; Dennis L,Murphy ; Stanley I,Rapoport

作者单位： Brain Physiology and Metabolism Section, National Institute on Aging, National Institutes of Health, Building 9, Room 1S128, Bethesda, MD, 20892, USA. yqu1@prdus.jnj.com ^[1]

主题词苯丙胺类(Amphetamines);动物(Animals);花生四烯酸(Arachidonic Acid);脑(Brain);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);磷脂酶A(Phospholipases A);磷脂酶类A2(Phospholipases A2);放射性核素显像(Radionuclide Imaging);受体, 血清素, 5-HT2A(Receptor, Serotonin, 5-HT2A);受体, 血清素, 5-HT2C(Receptor, Serotonin, 5-HT2C);血清素5-HT2受体激动剂(Serotonin 5-HT2 Receptor Agonists);血清素质膜转运蛋白质类(Serotonin Plasma Membrane Transport Proteins)

DOI 10.1007/s00213-005-2231-5

PMID 15834538

发布时间 2018-11-13

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Psychopharmacology

2005年180卷1期

12-20页

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5-HT2A/2C receptor signaling via phospholipase A2 and arachidonic acid is attenuated in mice lacking the serotonin reuptake transporter.

Psychopharmacology

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Psychopharmacology

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5-HT2A/2C receptor signaling via phospholipase A2 and arachidonic acid is attenuated in mice lacking the serotonin reuptake transporter.

Psychopharmacology

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Psychopharmacology

相关期刊

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