换一批CaMKIIalpha enhances the desensitization of NR2B-containing NMDA receptors by an autophosphorylation-dependent mechanism.
第一作者：
Suzanne,Sessoms-Sikes
第一单位：
Department of Molecular Physiology and Biophysics, The Center for Molecular Neuroscience, Vanderbilt University School of Medicine, Nashville, TN 37232, USA.
作者：
医学主题词
钙(Calcium);钙-钙调素依赖性蛋白激酶2型(Calcium-Calmodulin-Dependent Protein Kinase Type 2);钙-钙调素依赖性蛋白激酶类(Calcium-Calmodulin-Dependent Protein Kinases);细胞系(Cell Line);电生理学(Electrophysiology);人类(Humans);肾(Kidney);诱变(Mutagenesis);神经元可塑性(Neuronal Plasticity);磷酰化(Phosphorylation);RNA剪接(RNA Splicing);受体, N-甲基-D-天冬氨酸(Receptors, N-Methyl-D-Aspartate);信号传导(Signal Transduction);转染(Transfection)
DOI
10.1016/j.mcn.2005.01.006
PMID
15866054
发布时间
2015-08-13
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