Centromere protein H is up-regulated in primary human colorectal cancer and its overexpression induces aneuploidy.

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作者： Takeshi,Tomonaga ^[1] ; Kazuyuki,Matsushita ; Masumi,Ishibashi ; Masahiko,Nezu ; Hideaki,Shimada ; Takenori,Ochiai ; Kinya,Yoda ; Fumio,Nomura

作者单位： Department of Molecular Diagnosis (F8), Graduate School of Medicine, Chiba University, Inohana, Chuo-ku, Chiba, Japan. tomonaga@faculty.chiba-u.jp ^[1]

主题词 3T3细胞(3T3 Cells);非整倍性(Aneuploidy);动物(Animals);染色体不稳定性(Chromosomal Instability);染色体蛋白质类, 非组蛋白(Chromosomal Proteins, Non-Histone);结直肠肿瘤(Colorectal Neoplasms);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);小鼠(Mice);转染(Transfection);增量调节(Up-Regulation)

DOI 10.1158/0008-5472.CAN-04-3613

PMID 15930286

发布时间 2006-11-15

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Cancer research

2005年65卷11期

4683-9页

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Centromere protein H is up-regulated in primary human colorectal cancer and its overexpression induces aneuploidy.

Cancer research

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Cancer research

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Centromere protein H is up-regulated in primary human colorectal cancer and its overexpression induces aneuploidy.

Cancer research

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Cancer research

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