Induction of proinflammatory mediators requires activation of the TRAF, NIK, IKK and NF-kappaB signal transduction pathway in astrocytes infected with Escherichia coli.
第一作者:
J M,Kim
第一单位:
Department of Microbiology and Institute of Biomedical Science, Hanyang University College of Medicine, Seoul, Korea. jungmogg@hanyang.ac.kr
作者:
医学主题词
星形细胞(Astrocytes);细胞系, 肿瘤(Cell Line, Tumor);趋化因子CCL2(Chemokine CCL2);趋化因子CXCL1(Chemokine CXCL1);炎症趋化因子类(Chemokines);趋化因子, CXC(Chemokines, CXC);大肠杆菌感染(Escherichia coli Infections);人类(Humans);I-κB激酶(I-kappa B Kinase);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);白细胞介素8(Interleukin-8);NF-κB(NF-kappa B);一氧化氮合酶(Nitric Oxide Synthase);一氧化氮合酶Ⅱ型(Nitric Oxide Synthase Type II);磷酰化(Phosphorylation);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);信号传导(Signal Transduction);TNF受体相关因子2(TNF Receptor-Associated Factor 2);TNF受体相关因子5(TNF Receptor-Associated Factor 5);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1111/j.1365-2249.2005.02804.x
PMID
15932506
发布时间
2021-12-03
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