Breast cancer cell-targeted oxidative stress: enhancement of cancer cell uptake of conjugated linoleic acid, activation of p53, and inhibition of proliferation.

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作者： Craig D,Albright ^[1] ; Erich,Klem ; Anish A,Shah ; Patricia,Gallagher

作者单位： Department of Nutrition, McGavran-Greenberg Building, School of Public Health and School of Medicine, University of North Carolina, Chapel Hill, NC 27599-7461, USA. cdalbrig@email.unc.edu ^[1]

主题词乳腺肿瘤(Breast Neoplasms);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);DNA结合蛋白质类(DNA-Binding Proteins);电泳, 聚丙烯酰氨凝胶(Electrophoresis, Polyacrylamide Gel);女(雌)性(Female);荧光抗体技术(Fluorescent Antibody Technique);叉头转录因子类(Forkhead Transcription Factors);人类(Humans);免疫印迹法(Immunoblotting);亚油酸类, 共轭(Linoleic Acids, Conjugated);脂质过氧化作用(Lipid Peroxidation);氧化性应激(Oxidative Stress);转录因子(Transcription Factors);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53)

DOI 10.1016/j.yexmp.2005.05.005

PMID 15992797

发布时间 2016-11-24

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Experimental and molecular pathology

2005年79卷2期

118-25页

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Breast cancer cell-targeted oxidative stress: enhancement of cancer cell uptake of conjugated linoleic acid, activation of p53, and inhibition of proliferation.

Experimental and molecular pathology

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Experimental and molecular pathology

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Breast cancer cell-targeted oxidative stress: enhancement of cancer cell uptake of conjugated linoleic acid, activation of p53, and inhibition of proliferation.

Experimental and molecular pathology

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Experimental and molecular pathology

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