换一批
换一批Hyper-IgE syndrome is not associated with defects in several candidate toll-like receptor pathway genes.
第一作者：
Thomas R,Hawn
第一单位：
University of Washington School of Medicine, Seattle, WA 98103-8904, USA. thawn@u.washington.edu
作者：
主题词
衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);成年人(Adult);抗原, 分化(Antigens, Differentiation);细菌(Bacteria);女(雌)性(Female);人类(Humans);干扰素γ(Interferon-gamma);白细胞介素类(Interleukins);脂多糖类(Lipopolysaccharides);男(雄)性(Male);中年人(Middle Aged);髓样分化因子88(Myeloid Differentiation Factor 88);肽聚糖(Peptidoglycan);多态现象, 遗传(Polymorphism, Genetic);受体, 免疫(Receptors, Immunologic);信号传导(Signal Transduction);TNF受体相关因子6(TNF Receptor-Associated Factor 6);磷壁酸类(Teichoic Acids);Toll样受体1(Toll-Like Receptor 1);Toll样受体2(Toll-Like Receptor 2);Toll样受体6(Toll-Like Receptor 6);Toll样受体(Toll-Like Receptors);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);酵母多糖(Zymosan)
DOI
10.1016/j.humimm.2005.04.009
PMID
16112032
发布时间
2013-11-21
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Human immunology
842-7页
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