换一批
换一批A "traffic control" role for TGFbeta3: orchestrating dermal and epidermal cell motility during wound healing.
第一作者：
Balaji,Bandyopadhyay
第一单位：
Department of Dermatology and Norris Comprehensive Cancer Center, University of Southern California Keck School of Medicine, Los Angeles, CA 90033, USA.
作者：
主题词
激活素受体,Ⅰ型(Activin Receptors, Type I);抗体(Antibodies);细胞运动(Cell Movement);细胞, 培养的(Cells, Cultured);培养基, 无血清(Culture Media, Serum-Free);真皮(Dermis);内皮细胞(Endothelial Cells);表皮(Epidermis);成纤维细胞(Fibroblasts);基因表达(Gene Expression);人类(Humans);角蛋白细胞(Keratinocytes);黑素细胞(Melanocytes);突变(Mutation);磷酰化(Phosphorylation);血浆(Plasma);蛋白聚糖类(Proteoglycans);RNA, 小分子干扰(RNA, Small Interfering);受体, 转化生长因子β(Receptors, Transforming Growth Factor beta);血清(Serum);皮肤(Skin);Smad2蛋白质(Smad2 Protein);Smad3蛋白质(Smad3 Protein);转染(Transfection);转化生长因子β(Transforming Growth Factor beta);转化生长因子β3(Transforming Growth Factor beta3);伤口愈合(Wound Healing)
DOI
10.1083/jcb.200507111
PMID
16549496
发布时间
2023-11-05
- 浏览58
The Journal of cell biology
1093-105页
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