U(S)3 and U(S)3.5 protein kinases of herpes simplex virus 1 differ with respect to their functions in blocking apoptosis and in virion maturation and egress.
第一作者:
Alice P W,Poon
第一单位:
The Marjorie B. Kovler Viral Oncology Laboratories, The University of Chicago, 910 East 58th Street, Chicago, Illinois 60637, USA.
作者:
医学主题词
动物(Animals);细胞凋亡(Apoptosis);细胞系(Cell Line);细胞核(Cell Nucleus);环AMP依赖性蛋白激酶类(Cyclic AMP-Dependent Protein Kinases);疱疹病毒1型, 人(Herpesvirus 1, Human);组蛋白脱乙酰基酶2(Histone Deacetylase 2);组蛋白脱乙酰基酶类(Histone Deacetylases);人类(Humans);即早蛋白质类(Immediate-Early Proteins);亮肽菌素类(Leupeptins);核蛋白质类(Nuclear Proteins);磷酰化(Phosphorylation);蛋白质加工, 转译后(Protein Processing, Post-Translational);兔(Rabbits);阻遏蛋白质类(Repressor Proteins);病毒蛋白质类(Viral Proteins);病毒调节和辅助蛋白质类(Viral Regulatory and Accessory Proteins);病毒粒子(Virion);bcl相关死亡蛋白质(bcl-Associated Death Protein)
DOI
10.1128/JVI.80.8.3752-3764.2006
PMID
16571792
发布时间
2021-12-03
- 浏览26
Journal of virology
3752-64页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文


换一批



