换一批
换一批Aspirin and PPAR-alpha activators inhibit monocyte chemoattractant protein-1 expression induced by high glucose concentration in human endothelial cells.
第一作者：
Elena,Dragomir
第一单位：
Institute of Cellular Biology and Pathology Nicolae Simionescu, Bucharest, 8, BP Hasdeu Street, PO Box 35-14, 79691-Bucharest, Romania. elena.dragomir@icbp.ro
作者：
主题词
消炎药, 非甾类(Anti-Inflammatory Agents, Non-Steroidal);阿司匹林(Aspirin);细胞系(Cell Line);趋化因子CCL2(Chemokine CCL2);氯贝丁酯(Clofibrate);剂量效应关系, 药物(Dose-Response Relationship, Drug);内皮细胞(Endothelial Cells);非诺贝特(Fenofibrate);基因表达调控(Gene Expression Regulation);葡萄糖(Glucose);人类(Humans);NF-κB(NF-kappa B);鎓类化合物(Onium Compounds);PPARα(PPAR alpha);过氧化酶体激增剂(Peroxisome Proliferators);原癌基因蛋白质c-jun(Proto-Oncogene Proteins c-jun);活性氧(Reactive Oxygen Species);甲苯磺酰苯丙氨酰氯甲酮(Tosylphenylalanyl Chloromethyl Ketone);转录因子RelA(Transcription Factor RelA)
DOI
10.1016/j.vph.2006.02.006
PMID
16600694
发布时间
2017-11-16
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Vascular pharmacology
440-9页
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