TGF-beta1-induced thrombospondin-1 expression through the p38 MAPK pathway is abolished by fluvastatin in human coronary artery smooth muscle cells.
作者:
主题词
成年人(Adult);细胞, 培养的(Cells, Cultured);冠状动脉再狭窄(Coronary Restenosis);冠状血管(Coronary Vessels);剂量效应关系, 药物(Dose-Response Relationship, Drug);酶抑制剂(Enzyme Inhibitors);脂肪酸类, 单不饱和(Fatty Acids, Monounsaturated);基因表达调控(Gene Expression Regulation);人类(Humans);羟甲基戊二酰基CoA还原酶抑制剂(Hydroxymethylglutaryl-CoA Reductase Inhibitors);咪唑类(Imidazoles);吲哚类(Indoles);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);肌, 平滑, 血管(Muscle, Smooth, Vascular);肌细胞, 平滑肌(Myocytes, Smooth Muscle);磷酰化(Phosphorylation);吡啶类(Pyridines);RNA, 信使(RNA, Messenger);凝血酶敏感蛋白1(Thrombospondin 1);转化生长因子β(Transforming Growth Factor beta);转化生长因子β1(Transforming Growth Factor beta1);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1016/j.vph.2006.03.002
PMID
16624629
发布时间
2018-12-01
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Vascular pharmacology
469-75页
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