Deletion of the SOCS3 gene in liver parenchymal cells promotes hepatitis-induced hepatocarcinogenesis.
第一作者:
Hisanobu,Ogata
第一单位:
Division of Molecular and Cellular Immunology, Medical Institute of Bioregulation, Graduate School of Medical Science, Kyushu University, Higashiku, Fukuoka, Japan.
作者:
主题词
成年人(Adult);老年人(Aged);动物(Animals);癌, 肝细胞(Carcinoma, Hepatocellular);伴刀豆球蛋白A(Concanavalin A);女(雌)性(Female);基因缺失(Gene Deletion);肝细胞(Hepatocytes);人类(Humans);免疫组织化学(Immunohistochemistry);肝肿瘤(Liver Neoplasms);男(雄)性(Male);小鼠(Mice);中年人(Middle Aged);RNA, 肿瘤(RNA, Neoplasm);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);细胞因子信号转导蛋白抑制因子(Suppressor of Cytokine Signaling Proteins)
DOI
10.1053/j.gastro.2006.04.025
PMID
16831601
发布时间
2016-11-24
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Gastroenterology
179-93页
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