On the mechanisms of 12-O-tetradecanoylphorbol-13-acetate-induced growth arrest in pancreatic cancer cells.
作者:
主题词
细胞分裂(Cell Division);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);细胞周期蛋白A(Cyclin A);细胞周期蛋白B(Cyclin B);细胞周期蛋白B1(Cyclin B1);细胞周期蛋白E(Cyclin E);周期素依赖激酶抑制剂p21(Cyclin-Dependent Kinase Inhibitor p21);类黄酮物质(Flavonoids);G2期(G2 Phase);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);吲哚类(Indoles);MAP激酶激酶激酶类(MAP Kinase Kinase Kinases);马来酰亚胺类(Maleimides);丝裂原活化蛋白激酶1(Mitogen-Activated Protein Kinase 1);丝裂原活化蛋白激酶3(Mitogen-Activated Protein Kinase 3);胰腺肿瘤(Pancreatic Neoplasms);磷酰化(Phosphorylation);蛋白激酶C(Protein Kinase C);蛋白激酶抑制剂(Protein Kinase Inhibitors);RNA, 信使(RNA, Messenger);十四酰佛波乙酯(Tetradecanoylphorbol Acetate)
DOI
10.1097/01.mpa.0000226896.93945.41
PMID
16868480
发布时间
2018-08-15
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Pancreas
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