换一批
换一批Bcr-Abl signaling through the PI-3/S6 kinase pathway inhibits nuclear translocation of the transcription factor Bach2, which represses the antiapoptotic factor heme oxygenase-1.
作者：
主题词
主动转运, 细胞核(Active Transport, Cell Nucleus);细胞凋亡(Apoptosis);碱性亮氨酸拉链转录因子类(Basic-Leucine Zipper Transcription Factors);细胞系(Cell Line);融合蛋白质类, bcr-abl(Fusion Proteins, bcr-abl);血红素加氧酶-1(Heme Oxygenase-1);人类(Humans);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);蛋白质加工, 转译后(Protein Processing, Post-Translational);阻遏蛋白质类(Repressor Proteins);核糖体蛋白质S6激酶类(Ribosomal Protein S6 Kinases);信号传导(Signal Transduction)
DOI
10.1182/blood-2005-12-040972
PMID
17018862
发布时间
2021-02-06
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Blood
1211-9页
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