Differential roles of the co-stimulatory molecules GITR and CTLA-4 in the immune response to Trichinella spiralis.
作者:
主题词
动物(Animals);抗体, 蠕虫(Antibodies, Helminth);抗原, CD(Antigens, CD);抗原, 分化(Antigens, Differentiation);CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);女(雌)性(Female);流式细胞术(Flow Cytometry);糖皮质激素类诱导TNFR相关蛋白质(Glucocorticoid-Induced TNFR-Related Protein);免疫球蛋白E(Immunoglobulin E);免疫球蛋白G(Immunoglobulin G);免疫球蛋白M(Immunoglobulin M);白细胞介素10(Interleukin-10);白细胞介素4(Interleukin-4);肠(Intestines);淋巴结(Lymph Nodes);淋巴细胞亚群(Lymphocyte Subsets);小鼠(Mice);肌, 骨骼(Muscle, Skeletal);受体, 神经生长因子类(Receptors, Nerve Growth Factor);受体, 肿瘤坏死因子(Receptors, Tumor Necrosis Factor);脾(Spleen);旋毛线虫(Trichinella spiralis);旋毛虫病(Trichinellosis);增量调节(Up-Regulation)
DOI
10.1016/j.micinf.2006.08.013
PMID
17045510
发布时间
2016-10-20
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Microbes and infection
2803-10页
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