换一批Pertussis toxin is superior to TLR ligands in enhancing pathogenic autoimmunity, targeted at a neo-self antigen, by triggering robust expansion of Th1 cells and their cytokine production.
第一作者：
Chiaki,Fujimoto
第一单位：
Laboratory of Immunology, National Eye Institute, National Institute of Health, Bethesda, MD 20892-1857, USA.
作者：
医学主题词
动物(Animals);抗体, 封闭(Antibodies, Blocking);自身抗原(Autoantigens);自身免疫疾病(Autoimmune Diseases);细胞黏附分子(Cell Adhesion Molecules);细胞分化(Cell Differentiation);细胞因子类(Cytokines);干扰素γ(Interferon-gamma);白细胞介素12(Interleukin-12);晶体疾病(Lens Diseases);配体(Ligands);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);胞壁质酶(Muramidase);百白咳毒素(Pertussis Toxin);Th1细胞(Th1 Cells);Toll样受体(Toll-Like Receptors)
DOI
10.4049/jimmunol.177.10.6896
PMID
17082604
发布时间
2019-05-16
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