Extracellular matrix fibronectin increases prostaglandin E2 receptor subtype EP4 in lung carcinoma cells through multiple signaling pathways: the role of AP-2.
作者:
主题词
癌(Carcinoma);细胞系, 肿瘤(Cell Line, Tumor);地诺前列酮(Dinoprostone);酶抑制剂(Enzyme Inhibitors);细胞外基质(Extracellular Matrix);纤连蛋白类(Fibronectins);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);人类(Humans);整合素α5β1(Integrin alpha5beta1);肺肿瘤(Lung Neoplasms);模型, 生物学(Models, Biological);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);RNA, 小分子干扰(RNA, Small Interfering);受体, 前列腺素E(Receptors, Prostaglandin E);受体, 前列腺素E, EP4亚型(Receptors, Prostaglandin E, EP4 Subtype);信号传导(Signal Transduction);转录因子AP-2(Transcription Factor AP-2)
DOI
10.1074/jbc.M610308200
PMID
17237224
发布时间
2021-02-09
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