Reduced CCR4, interleukin-13 and GATA-3 up-regulation in response to type 2 cytokines of cord blood T lymphocytes in infants at genetic risk of type 1 diabetes.
第一作者:
Kristiina,Luopajärvi
第一单位:
Hospital for Children and Adolescents, University of Helsinki, Helsinki, Finland. kristiina.luopajarvi@hus.fi
作者:
主题词
细胞极性(Cell Polarity);细胞, 培养的(Cells, Cultured);细胞因子类(Cytokines);糖尿病, 1型(Diabetes Mellitus, Type 1);胎血(Fetal Blood);GATA3转录因子(GATA3 Transcription Factor);疾病遗传易感性(Genetic Predisposition to Disease);HLA-DQ抗原(HLA-DQ Antigens);HLA-DR4抗原(HLA-DR4 Antigen);组织相容性试验(Histocompatibility Testing);人类(Humans);婴儿, 新生(Infant, Newborn);白细胞介素13(Interleukin-13);RNA, 信使(RNA, Messenger);受体, CCR4(Receptors, CCR4);受体, CXCR4(Receptors, CXCR4);受体, 趋化因子(Receptors, Chemokine);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);T淋巴细胞亚群(T-Lymphocyte Subsets);增量调节(Up-Regulation)
DOI
10.1111/j.1365-2567.2007.02557.x
PMID
17244154
发布时间
2018-11-13
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Immunology
189-96页
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