换一批Prolonged shear stress and KLF2 suppress constitutive proinflammatory transcription through inhibition of ATF2.
第一作者：
Joost O,Fledderus
第一单位：
Department of Medical Biochemistry, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands.
作者：
医学主题词
转录激活因子2(Activating Transcription Factor 2);动脉粥样硬化(Atherosclerosis);细胞核(Cell Nucleus);细胞, 培养的(Cells, Cultured);聚类分析(Cluster Analysis);内皮细胞(Endothelial Cells);基因表达调控(Gene Expression Regulation);人类(Humans);炎症(Inflammation);Kruppel样转录因子类(Kruppel-Like Transcription Factors);磷酰化(Phosphorylation);蛋白激酶类(Protein Kinases);RNA, 小分子干扰(RNA, Small Interfering);应力, 物理(Stress, Mechanical);时间因素(Time Factors);转录, 遗传(Transcription, Genetic);肿瘤坏死因子α(Tumor Necrosis Factor-alpha)
DOI
10.1182/blood-2006-07-036020
PMID
17244683
发布时间
2021-02-06
- 浏览19
Blood
4249-57页
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