换一批
换一批Cellular demise and inflammatory microglial activation during beta-amyloid toxicity are governed by Wnt1 and canonical signaling pathways.
作者：
主题词
淀粉样β肽类(Amyloid beta-Peptides);动物(Animals);细胞凋亡(Apoptosis);细胞系, 肿瘤(Cell Line, Tumor);细胞膜(Cell Membrane);细胞增殖(Cell Proliferation);DNA损伤(DNA Damage);基因沉默(Gene Silencing);基因组, 人(Genome, Human);糖原合成酶激酶3(Glycogen Synthase Kinase 3);人类(Humans);炎症(Inflammation);小神经胶质细胞(Microglia);神经保护药(Neuroprotective Agents);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷脂酰丝氨酸类(Phosphatidylserines);磷酰化(Phosphorylation);蛋白质转运(Protein Transport);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);信号传导(Signal Transduction);Wnt1蛋白质(Wnt1 Protein);β连环素(beta Catenin)
DOI
10.1016/j.cellsig.2006.12.009
PMID
17289346
发布时间
2022-03-16
- 浏览15
Cellular signalling
1150-62页
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