换一批
换一批TRAF-1, -2, -3, -5, and -6 are induced in atherosclerotic plaques and differentially mediate proinflammatory functions of CD40L in endothelial cells.
第一作者：
Andreas,Zirlik
第一单位：
Donald W. Reynolds Cardiovascular Research Center, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 77 Avenue Louis Pasteur, Boston, MA 02115, USA.
作者：
主题词
动物(Animals);动脉粥样硬化(Atherosclerosis);印迹法, 蛋白质(Blotting, Western);CD40配体(CD40 Ligand);疾病恶化(Disease Progression);内皮细胞(Endothelial Cells);内皮, 血管(Endothelium, Vascular);基因表达(Gene Expression);人类(Humans);炎症(Inflammation);小鼠(Mice);聚合酶链反应(Polymerase Chain Reaction);RNA, 小分子干扰(RNA, Small Interfering);隐静脉(Saphenous Vein);TNF受体相关因子1(TNF Receptor-Associated Factor 1);TNF受体相关因子2(TNF Receptor-Associated Factor 2);TNF受体相关因子3(TNF Receptor-Associated Factor 3);TNF受体相关因子5(TNF Receptor-Associated Factor 5);TNF受体相关因子6(TNF Receptor-Associated Factor 6)
DOI
10.1161/ATVBAHA.107.140566
PMID
17332487
发布时间
2022-03-16
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