换一批Conditional deletion of focal adhesion kinase leads to defects in ventricular septation and outflow tract alignment.
第一作者：
Zeenat S,Hakim
第一单位：
Department of Pathology and Carolina Cardiovascular Biology Center, 501 Brinkhous-Bullitt Bldg. CB 7525, University of North Carolina, Chapel Hill, NC 27599, USA.
作者：
主题词
动物(Animals);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);细胞存活(Cell Survival);Crk相关底物(Crk-Associated Substrate Protein);胚胎, 哺乳动物(Embryo, Mammalian);女(雌)性(Female);黏着斑蛋白酪氨酸激酶类(Focal Adhesion Protein-Tyrosine Kinases);基因缺失(Gene Deletion);心脏缺损, 先天性(Heart Defects, Congenital);心室(Heart Ventricles);同源盒结构域蛋白质类(Homeodomain Proteins);男(雄)性(Male);小鼠(Mice);形态发生(Morphogenesis);肌细胞, 心脏(Myocytes, Cardiac);肌原纤维(Myofibrils);表型(Phenotype);磷酰化(Phosphorylation);转录因子(Transcription Factors)
DOI
10.1128/MCB.00068-07
PMID
17526730
发布时间
2025-05-29
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